New Study Reveals That All Covid Variants Have Been Created in BioLabs

According to a new pre-print published on August 5th 2023, a Japanese research team seems to find out that almost all Covid variants have been manufactured in biolabs.

By examining viral sequences discovered “in the wild” and stored in public databases, Atsuki Tanaka and Takayuki Miyazawa of Osaka Medical University and Kyoto University sought to track the historical evolution of the omicron version of SARS-CoV2.

In doing so, they discovered approximately 100 distinct omicron subvariants that were not possible to have developed naturally. These mutations appear to be unmistakable evidence of the widespread lab production and distribution of covid viruses.

Additionally, the variations seem to comprise extensive panels of mutations typical of those employed in “reverse genetics” investigations to methodically examine the characteristics of various virus sections.

In sequences from Puerto Rico that were deposited in databases in 2020, more than a year before the announcement of the finding of omicron in South Africa, the investigators also discovered exact matches to omicron variations.

Tanaka and Miyazawa contend that all variants appearing since the initial Wuhan outbreak are unnatural and hypothesize that they represent an experimental program to test factors influencing the infectivity and pathogenicity of SARS-CoV2 in the general population. These observations include observations of implausibly low numbers of “silent” mutations in SARS-CoV2 variants.

The study discusses the evolution of viruses and organisms, focusing on the concept of genetic information. SARS-CoV2 is a virus that uses RNA strings for information storage. The genetic material is divided into genes, which encode proteins. Proteins are active molecules synthesized by cells using the gene as a blueprint. The genetic sequence is copied and passed on to the next generation, but mutations can accumulate over time.

In order to understand the several general types of mutation and how they affect the organism, consider the following:

Many won’t have any impact. The majority of times, changing a single letter in DNA or RNA will not actually alter the sequence of the protein. Such “synonymous” mutations typically have no consequences, thus natural evolution just accumulates them over time. The absence of synonymous mutations, as we shall see, is a crucial indicator that the genesis of omicron and other variations is not spontaneous.

For the same reason that randomly changing a part of any working system is likely to cause it to malfunction, the vast majority of those that do have an effect will be detrimental. Such mutations will quickly vanish.

Rarely will a mutation result in a modification that is advantageous. As a result of creatures bearing these mutations surviving and reproducing more successfully, they will then spread throughout subsequent generations.

So, all life forms gradually develop mutations, some of which are silent and some of which are advantageous. That’s evolution.

This article focuses on Tanaka and Miyazawa’s study of the Spike protein in SARS-CoV2, specifically the gene encoding it. They used public databases to trace the evolutionary history of the omicron BA1 spike protein, aiming to answer the order in which these 37 mutations accumulate.

Tanaka and Miyazawa worked backwards by searching for sequences lacking just one of the 37 omicron mutations, assuming that one of them would match the last mutation in the series. They made a series of 37 database queries using sequences each lacking just one of the omicron BA1 mutations, with the hope that one of them would find a match, indicating the last mutation in the series.

The results showed that all but one of the 37 mutations were the most recent, making no material difference. The researchers’ brains must have exploded when they ran queries for each of the 37 mutations, which makes no material difference.

A panel of variants with individually-reversed omicron BA1 mutations cannot arise naturally. The figure below shows that for all but one mutation in the omicron sub-variant BA1, a strain exists in which the mutation is absent. This highlights the importance of understanding the evolution of the Spike protein and its role in the evolution of SARS-CoV2.

The study by Tanaka and Miyazawa explores the possibility of recombination in the evolution of SARS-CoV2 variants. They argue that recombination would require the presence of omicron BA1 viruses and other ancestral viruses in the same cell at the same time, which would be extremely rare due to the frequencies involved and the need for creating so many reversion mutations.

Explaining these reversions by recombination would require a section of RNA in omicron BA1 containing the mutation to be reversed, which would have to be cleanly swapped, such that the mutations to either side were unaffected. Cross-overs between the variants would have to be aligned of a stretch of common sequence between the two strains, but this is not possible for some mutations. Recombination would also leave marks in the flanking regions of the virus either side of the spike protein gene, which were not found.

Tanaka and Miyazawa found further evidence that raises fundamental questions about omicron’s history. They conducted database queries to look for signs that recombination had been involved, finding 29 variants attributed to Puerto Rico that exactly match either omicron BA1 or BA2, based on spike protein sequences. These sequences were deposited in the database in 2020, over a year before detection of omicron in South Africa was announced in November 2021.

The lack of “synonymous” mutations strongly suggests artificial origins of SARS-CoV2 variants. The authors argue that natural evolution would always create neutral synonymous mutations at a greater rate than non-synonymous mutations, which can only persist if they result in a design improvement in the protein they encode. This implausible observation is not limited to omicron, as there were no synonymous mutations in the Alpha, Beta, Gamma, Delta, or Mu variants, but only one each in the Lambda and Omicron variants.

The presence of almost complete panels of individual reversions of every mutation in three omicron lineages cannot plausibly be natural. Instead, it looks like a systematic exercise in reverse genetics to test the effects of each omicron mutation on the virus’s behavior. The lack of synonymous mutations in other variants suggests that all variants described after the original Wuhan strain have artificial origins.

The authors suggest that the variants they have found are part of an experiment to characterize the spike protein and the effects of mutations on the virus’ behavior. The lack of findings to date that many of the various mutations seen, especially in the early variants, are associated with increased viral infection supports the hypothesis that each variant was artificially synthesized to identify the amino acids of the S protein responsible for infectivity and pathogenicity. If the observations and inferences in this paper are correct, they provide indisputable evidence that the entire history of SARS-CoV2, at least subsequent to the emergence of the original strain, is artificial.

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